Pseudomonas example

A search in ISI, using pseudomonas, restricting to title and Review articles: gets 136. Then find something that W&L has:
Govan JRW, Deretic V
Microbial pathogenesis in cystic fibrosis: Mucoid Pseudomonas aeruginosa and Burkholderia cepacia
MICROBIOL REV 60: (3) 539-& SEP 1996

Eaton RW
p-Cumate catabolic pathway in Pseudomonas putida F1: Cloning and characterization of DNA carrying the cmt operon
J BACTERIOL 178: (5) 1351-1362 MAR 1996

DERETIC V, SCHURR MJ, BOUCHER JC, et al.
CONVERSION OF PSEUDOMONAS-AERUGINOSA TO MUCOIDY IN CYSTIC-FIBROSIS - ENVIRONMENTAL-STRESS AND REGULATION OF BACTERIAL VIRULENCE BY ALTERNATIVE SIGMA-FACTORS
J BACTERIOL 176: (10) 2773-2780 MAY 1994

OSULLIVAN DJ, OGARA F
TRAITS OF FLUORESCENT PSEUDOMONAS SPP INVOLVED IN SUPPRESSION OF PLANT-ROOT PATHOGENS
MICROBIOL REV 56: (4) 662-676 DEC 1992

Not much to work with, but maybe a start. Here's the abstract for the first one, which cites 506 sources, doubtless mostly from the medical literature:
Abstract:
Respiratory infections with Pseudomonas aeruginosa and Burkholderia cepacia play a major role in the pathogenesis of cystic fibrosis (CF). This review summarizes the latest advances in understanding host-pathogen interactions in CF with an emphasis on the role and control of conversion to mucoidy in P. aeruginosa a phenomenon epitomizing the adaptation of this opportunistic pathogen to the chronic chourse of infection in CF, and on the innate resistance to antibiotics of B. cepacia, person-to-person spread, and sometimes rapidly fatal disease caused by this organism. While understanding the mechanism of conversion to mucoidy in P. aeruginosa has progressed to the point where this phenomenon has evolved into a model system for studying bacterial stress response in microbial pathogenesis, the more recent challenge with B. cepacia, which has emerged as a potent bona fide CF pathogen, is discussed in the context of clinical issues, taxonomy, transmission, and potential modes of pathogenicity.

KeyWords Plus:
TUMOR-NECROSIS-FACTOR, CHRONIC GRANULOMATOUS-DISEASE, O-SIDE-CHAINS, ANTI-SIGMA FACTOR, RESPIRATORY EPITHELIAL-CELLS, CONTROLS ALGINATE STRUCTURE, BIOSYNTHETIC GENE-CLUSTER, STRESS-RESPONSE PROTEIN, SERUM IGG RESPONSE, BURNED-MOUSE MODEL

One of the cited references is from Nature: 
DEFECTIVE ACIDIFICATION OF INTRACELLULAR ORGANELLES IN CYSTIC-FIBROSIS
      BARASCH J, KISS B, PRINCE A, SAIMAN L, GRUENERT D, ALAWQATI Q
                                        NATURE 
                                 352: (6330) 70-73 JUL 4 1991
Another from J Exp Biol: 
CHLORIDE CHANNELS OF INTRACELLULAR ORGANELLES AND THEIR POTENTIAL ROLE IN
                                    CYSTIC-FIBROSIS
                            ALAWQATI Q, BARASCH J, LANDRY D
                           JOURNAL OF EXPERIMENTAL BIOLOGY 
                                   172: 245-266 NOV 1992
two from J Bacteriology: 
HIGH OSMOLARITY IS A SIGNAL FOR ENHANCED ALGD TRANSCRIPTION IN MUCOID AND
                    NONMUCOID PSEUDOMONAS-AERUGINOSA STRAINS
                         BERRY A, DEVAULT JD, CHAKRABARTY AM
                               JOURNAL OF BACTERIOLOGY 
                                 171: (5) 2312-2317 MAY 1989

and

Two distinct loci affecting conversion to mucoidy in Pseudomonas 
aeruginosa in cystic fibrosis encode homologs of the serine protease HtrA
        Boucher JC, MartinezSalazar J, Schurr MJ, Mudd MH, Yu H, Deretic V
                               JOURNAL OF BACTERIOLOGY 
                                  178: (2) 511-523 JAN 1996
(there are others as well)

Starting from this base, it seems that I could work on cystic fibrosis --take that disease as my topic, and use the available resources to put together a short bibliography summarizing my discoveries.

My next step was to do a search for 'pseudomonas' in article titles in Science magazine, and here's what I got: 

Role of Mutant CFTR in Hypersusceptibility of Cystic Fibrosis Patients to 
Lung Infections 
              Science 1996 January 5; 271 (5245):64 (in Reports)
              G. B. Pier, M. Grout, T. S. Zaidi, J. C. Olsen, L. G. 
              Johnson, J. R. Yankaskas, J. B. Goldberg

             
             
Molecular Basis of Gene-for-Gene Specificity in Bacterial 
Speck Disease of Tomato 
              Science 1996 December 20; 274 (5295):2063 (in Reports)
              S. R. Scofield, C. M. Tobias, J. P. Rathjen, J. H. Chang, 
              D. T. Lavelle, R. W Michelmore, B. J. Staskawicz

             
             
Initiation of Plant Disease Resistance by Physical 
Interaction of AvrPto and Pto Kinase 
              Science 1996 December 20; 274 (5295):2060 (in Reports)
              X. Tang, R. D. Frederick, J. Zhou, D. A. Halterman, Y. Jia, 
              G. B. Martin

             
DOE Serves Up Bioremediation Funds 
              Science 1996 November 15; 274 (5290):1071 (in ScienceScope)


Denied clearance? 
              Science 1996 January 5; 271 (5245):9 (in This Week in Science)

             
Crystal Structure of the Biphenyl-Cleaving Extradiol 
Dioxygenase from a PCB-Degrading Pseudomonad 
              Science 1995 November 10; 270 (5238):976 (in Reports)
              S. Han, L. D. Eltis, K. N. Timmis, S. W. Muchmore, J. T. Bolin
I don't know what good these are without retrieving them --though some have nothing to do with cystic fibrosis. The "Denied Clearance" one looks worthwhile, and leads to this: 
Role of Mutant CFTR in Hypersusceptibility of Cystic Fibrosis Patients to
Lung Infections

Gerald B. PierJ(1),J Martha Grout,J Tanweer S. Zaidi,J John C. Olsen,J 
Larry G. Johnson,J James R. Yankaskas,J
 
(5 Jan 1996 pp 64-67)
So if I needed to inform myself about cystic fibrosis I have a lot of tools to choose amongst, including